medigraphic.com
Anales de Otorrinolaringología Mexicana

2005, Number 2

<< Back Next >>

Otorrinolaringología 2005; 50 (2)

Parents age and hypoacusia-prelingual deafness

Peñaloza-López YR, García-Pedroza F, Castillo-Maya G

Language: Spanish
References: 33
Page: 40-48
PDF size: 72.05 Kb.


ABSTRACT

Objective. To analyze the distribution by age of the ancestors of seven patients with hypoacusia-prelingual deafness (H-PD) attributed to bilateral cortipathies, in addition to a group of congenital malformations of external ear (CMEE). Material and methods. The parents of the studied patients were distributed by age in three groups (younger than 20 years, between 20 and 35 years, and 35 or more years). The results were compared with the reported by the Instituto Nacional de Geografía, Estadística e Informática (INEGI) of Mexico. Results. Of 5 614 files of Instituto Nacional de la Comunicación Humana (INCH), 708 corresponded to H-PD. Most frequent cortipathies were: 31.8% by nondetermined cause, 23.2% related to adverse perinatal factors, 13.8% of hereditary type, and 10.7% by infections. In relation to the three ancestors etary groups participant in the study, the percentage differences were analyzed respect to the ages of the general population. Conclusions. It turns out controversial to establish relations between H-PD and age of the ancestors. The studied groups of ages reflect differences that express genetic, social and environmental context in that ancestors and patients evolve. In this work, the possible relation between these factors and the differences found when comparing the ages of ancestors of patients with H-PD and the ages of the general population is examined.


REFERENCES

  1. Peñaloza Y, Reyna V, Poblano A. Detección temprana de sordera prelingüística. Bol Méd Hosp Infant Méx 1988; 45: 155-9.

  2. García-Pedroza F, Peñaloza Y, Poblano A. La sordera congénita en México. Bol Méd Hosp Infant Méx 2000; 57: 633-40.

  3. Castillo-Maya G, Peñaloza Y, Hernández-Orozco F. Etiología de la hipoacusia–sordera. Gac Méd Méx 2001; 137: 541-8.

  4. Van Camp G. Hereditary hearing loss. (http://www.uia.ac.be/dnalab/hhh).

  5. Cryns K, Van Camp G. Deafness genes and their diagnostic applicatons. Audiol Neurootol 2004; 9 (1): 2-22.

  6. Marion R. The genetic anatomy of hearing: a clinician’s view. En: Genetics of hearing impairment. R Ruben Sciences, 1991; 630: 32-7.

  7. Lindsay JR. Profound childhood deafness. Inner ear pathology. Ann Otol Laryngol 1973; 82 (Suppl 5): 1-121.

  8. Solórzano F, López-Álvarez A, Muñoz MT, et al. Síndrome de rubéola congénita en lactantes atendidos en un hospital pediátrico. Gac Med Méx 2001; 137 (2): 105-9.

  9. Dennis JM, Neely JG. Otoneurologic diseases and associated audiologic profiles. En: Diagnostic audiology. Northern J Pro-Ed, 1990.

  10. Hicks T, Fowler K, Richardson M, et al. Congenital cytomegalovirus infection and neonatal auditory screening. J Pediatr 1993; 23 (5): 779-82.

  11. Cremers CW, Van Rijn PM. Acquired causes of deafness in childhood. Ann N York Acad Sci 1991; 630: 197-202.

  12. Joint Committee on Infant Hearing Year 2000. Position statement: principles and guidelines for early detection and intervention programs. (www.infanthearing.org/jcih/)

  13. Kelly LE. Adolescent mothers: what factors relate to level of preventive health care sought for their infants? J Pediatr Nurs 1995; 10 (2): 105-13.

  14. Folsom RC, Widen JE, Vohr BR, et al. Multicenter consortium on identification of neonatal hearing impairment: recruitment and follow-up. Ear Hear 2000; 21 (5): 462-70.

  15. Croen LA, Shaw GM. Young maternal age and congenital malformations: a population-based study. Am J Public Health 1995; 85 (5): 710-5.

  16. Parving A, Dafydd S. Profound permanent hearing impairment in childhood: causative factors in two European countries. Acta Otolaryngol (Stockh) 1997; 117: 158-60.

  17. Medina P. Diagnóstico prenatal. En: Genética clínica. El Manual Moderno, México, 1994.

  18. 18 Nazer J, Cifuentes L, Ruiz G, Pizarro MT. Maternal age as a risk factor for congenital malformations. Rev Med Child 1994; 122 (3): 299-303.

  19. Sutton GJ, Rowe SJ. Risk factors for childhood sensorineural hearing loss in the Oxford region. Br J Audiol 1997; 31 (1): 39-54.

  20. Epstein CHJ. Down syndrome (trisomy 21). En: The metabolic and molecular bases of inherited disease. McGraw-Hill, 2001.

  21. Tellier AL, Cormier-Daire V, Abadie V, et al. CHARGE syndrome: report of 47 cases and review. Am J Med Genet 1998; 76 (5): 402-9.

  22. Castilla EE, Orioli IM. Prevalence rates of microtia in South America. Int J Epidemiol 1986; 15 (3): 364-8.

  23. Fisch L. Parenteral age birth order and congenital deafness. Audiology 1971; 10: 284-90.

  24. Mutchinick O, Lisker R, Babinsky V. Programa Mexicano de Registro y Vigilancia Epidemiológica de Malformaciones Congénitas Externas. Salud Pública de México 1988; 30 (1): 88-100.

  25. Guízar J. Genética clínica. El Manual Moderno, México, 1994.

  26. Musiek F, Rintelmann W. Contemporary perspectives in hearing assessment. Allyn & Bacon, 1999.

  27. Jacobson J, Northern J. Diagnostic audiology. Pro-Ed, Austin (Tx), 1990.

  28. Estadísticas demográficas. Cuaderno de población No. 7. Instituto Nacional de Estadística Geografía e Informática, México, 1996.

  29. Mortalidad 1999. Secretaría de Salud. Julio de 2001. Principales causas de mortalidad infantil en l999.

  30. Cortés A, Valdés C. Se registran 370 mil partos sin atención al año: SSA. El Universal, 21 de febrero de 2002.

  31. Baird PA, Sadovnick AD, Yee IM. Maternal age and birth defects: a population study. Lancet 1991; 337 (8740): 527-30.

  32. Lina-Granade G, Collet L, Morgon A. Physiopathological investigations in a family with history of unilateral hereditary deafness. Acta Otolayngol (Stock) 1995; 115 (2): 196-201.

  33. Hu DN, Qui WQ, Wu BT, et al. Genetic aspects of antibiotic induced deafness: mitochondrial inheritance. J Med Genet 1991; 28: 79-83.




2020     |     www.medigraphic.com