2000, Number 5
<< Back Next >>
Cir Cir 2000; 68 (5)
Measurement of capillary fuge in pre-eclampsia-eclampsia
Briones-Garduño JC, Díaz León-Ponce M, Gómez Bravo-Topete E, Avila-Esquivel F, Ochoa Ruiz-Esparza C, Briones-Vega CG, Urrutia-Torres F
Language: Spanish
References: 12
Page: 194-197
PDF size: 75.90 Kb.
ABSTRACT
Introduction: We propose two parameters for capillary fuge estimation in preeclamptic-eclamptic patients as a result of alterations in Starling forces.
Objective: To report COPc and BI values in preeclamptic-eclamptic patients compared with normal pregnancies.
Method: In a prospective study we had revised COPc and EI in 87 preeclamptic/eclamptic patients (Group A) and compared them with 50 normal pregnancies (Group B) according to Landis-Pappenheimer formula and calculated COPc, as the result of the division of the COPc and MAP denominate BI.
Results: Group A had values of 15.3 ± 4.5 for the COPc and 0.11 ± 0.03 for the BI; Group B had values of 24.0 ± 4.4 for the COPc and 0.27 ± 0.03 for the BI. (p ‹ 0.005 and p = 0.300 respectively).
Conclusions: We observed a significant decrease of COPc and BI in preeclamptic-eclamptic patients, and we suggested capillary fuge quantitative indexes for capillary fuge.
REFERENCES
Díaz de León PM, Herrera RC, Cruz LC. Valor normal de la presión coloidosmótica en adultos sanos de la Ciudad de México. Arch Invest Med (Mex) 1981; 12: 307-321.
Ayala AA. Toxemia Gravídica. Un problema multidisciplinario. Gac Med Mex 1991; 127(3): 215-219.
Díaz de León PM, Briones GJC, Kably AA, Barrón VJ, Espinosa MML, Juárez AG. Medicina crítica en obstetricia. Rev Asoc Mex Med Crit Ter Int 1997; 2: 36-40.
Díaz de León PM, Espinosa MML, Briones GJC, López Llera MM, Briones VCG. Microangiopatía trombótica y hemólisis intravascular en la preeclampsia-eclampsia, los eslabones perdidos en el síndrome de HELLP. Rev Asoc Mex Med Crit Ter Int 1997; 1: 4-8.
Palma CP, Briones GJC, Molinar RF, Vázquez HMI, Baltazár TJA. Perfil hemodinámico en pacientes con preeclampsia severa y eclampsia. Rev Asoc Mex Med Crit Ter Int 1994; 1: 9-15.
Clark SL, Greenspoon JS, Aldahl DRN, Phelan JP. Severe preeclampsia with persistent oliguria: management of hemodynamic subsets. Am J Obstet Gynecol 1986; 154: 490-494.
Williams DJ, Swiet M. The pathophysiology of preeclampsia. Intensive Care Med 1997; 23: 620-629.
Briones GJC, Díaz de León PM, Castañón GJA, Briones VCG. Presión coloidosmótica (PCO) en el embarazo normal y puerperio fisiológico. Rev Asoc Mex Med Crit Ter Int 1997; 11(2): 45-47.
Dekker GA, Van Geijn HP. Endothelial dysfunction in preeclampsia. Part II: Reducing the adverse consequences of endothelial cell dysfunction in preeclampsia; therapeutic perspectives. J Perinat Med 1996; (24): 119-139.
Valenzuela GJ. Is an decrease in plasma oncotic pressure enough to explain the edema of pregnancy? Am J Obstet Gynecol 1989; 161: 1624-1627.
Briones GJC, Castañón GJA, Díaz de León PM, Madrazo NI, Deseano EJL, Briones VCG, Urrutia TF. Hemodinamia cerebral en embarazo normal y en preeclampsia-eclampsia. Rev Asoc Mex Med Crit Ter Int 1997; 11(4): 106-111.
Villegas CH. Ultraestructura de la placenta, In: Briones GJC, Díaz de León PM, Editores. Preeclampsia- eclampsia. México editorial. Distribuidora y Editora Mexicana, S.A. de C.V; 2000 p. 79 –90.