2002, Número 4
<< Anterior Siguiente >>
Ann Hepatol 2002; 1 (4)
Overview on the mechanisms of drug-induced liver cell death
Grattagliano I, Portincasa P, Palmieri VO, Palasciano G
Idioma: Ingles.
Referencias bibliográficas: 37
Paginas: 162-168
Archivo PDF: 105.73 Kb.
FRAGMENTO
Sin resumen
REFERENCIAS (EN ESTE ARTÍCULO)
Drug-induced hepatotoxicity. Handb Exp Pharm n. 121 Eds: Cameron RG, Feuer G, de la Iglesia FA. Springer-Verlag, Berlin-Heidelberg, 1996.
Farber E. Programmed cell death: necrosis versus apoptosis. Mod Pathol 1994; 7: 605-609.
Pessayre D, Larrey D. Acute and chronic drug-induced hepatitis. Bailliere’s Clin Gastroenterol 1988; 2: 385-423.
Lauterburg BH. Early disturbance of calcium translocation across the plasma membrane in toxic liver injury. Hepatology 1987; 7: 1179-1183.
Evans MA, Harbison RD. Cocaine-induced hepatotoxicity in mice. Toxicol Appl Pharmacol 1978; 45: 739-754.
Leist M, Gantner F, Kunstle G, et al. Cytokine-mediated hepatic apoptosis. Rev Physiol Biochem Pharmacol 1998; 133: 109-155.
Scaffidi G, Fulda S, Srinivasasn A, et al. Two CD95 (APO-1/Fas) signaling pathways. EMBO J 1998; 17: 1675-1687.
Lemaster JJ. V. Necrapoptosis and the mitochondrial permeability transition: shared pathways to necrosis and apoptosis. Am J Physiol 1999; 276: G1-G6.
Ashkenazi A, Dixit V. Death receptors: signaling and modulation. Science 1998; 281: 1305-1308.
Latta M, Kunstle G, Leist M, et al. Metabolic depletion of ATP by fructose inversely controls CD95-and tumor necrosis factor receptor 1-mediated hepatic apoptosis. J Exp Med 2000; 191: 1975-1985.
Hatano E, Bennett BL, Manning AM, et al. NF-kappaB stimulates inducible nitric oxide synthase to protect mouse hepatocytes from TNF-alpha- and Fas-mediated apoptosis. Gastroenterology 2001; 120: 1251-12161.
Colell A, Garcia-Ruiz C, Miranda M, et al. Selective glutathione depletion of mitochondria by ethanol sensitizes hepatocytes to tumor necrosis factor. Gastroenterology 1998; 115: 1541-1551.
Hampton MB, Orrenius S. Dual regulation of caspase activity by hydrogen peroxide: implications for apoptosis. FEBS Lett 1997; 414: 552-556.
Laskin DL, Gardner CR, Price VF, et al. Modulation of macrophage functioning abrogates the acute hepatotoxicity of acetaminophen. Hepatology 1995; 21: 1045-1050.
Grattagliano I, Palmieri VO, Palasciano G. Hepatotoxicity of polyunsaturated fatty acids in alcohol abuser. J Hepatol 2002; 37: 291-292.
Nelson SD. Molecular mechanisms of the hepatotoxicity caused by acetaminophen. Sem Liver Dis 1990; 10: 267-278.
Vendemiale G, Grattagliano I, Altomare E, et al. Effect of acetaminophen administration on hepatic glutathione compartmentation and mitochondrial energy metabolism in the rat. Biochem Pharmacol 1996; 52: 1147-1154.
Deleve LD, Kaplowitz N. Importance and regulation of hepatic glutathione. Sem Liver Dis 1990; 10: 251-266.
Rosser BG, Gores GJ. Liver cell necrosis: cellular mechanisms and clinical implications. Gastroenterology 1995; 108: 252-275.
Ray SD, Mumaw VR, Raje RR, et al. Protection of acetaminophen-induced hepatocellular apoptosis and necrosis by cholesteryl hemisuccinate pretreatment. J Pharmacol Exp Ther 1996; 279: 1470-1483.
Barros LF, Stutzin A, Calixto A, et al. Nonselective cation channels as effectors of free radical-induced rat liver cell necrosis. Hepatology 2001; 33: 114-122.
Bissel M, Gores GJ, Laskin DL, et al. Drug-induced liver injury: mechanisms and test systems. Hepatology 2001; 33: 1009-1013.
Anderson ME, Naganuma A, Meister A. Protection against cisplatin toxicity by administration of glutathione ester. FASEB J 1990; 4: 3251-3255.
Krahenbhul S. Mitochondria: important target for drug toxicity? J Hepatol 2001; 34: 334-336.
Berson A, Fau D, Fornacciari R, et al. Mechanisms for experimental buprenorphine hepatotoxicity: major role of mitochondrial dysfunction versus metabolic activation. J Hepatol 2001; 34: 261-269.
Susin SA, Zamzami N, Kroemer G. Mitochondria as regulators of apoptosis: doubt no more. Biochim Biophys Acta 1998; 1336: 151-165.
Hoouzi D, Lekehal M, Tinel M, et al. Prolonged, but not acute, glutathione depletion promotes Fas-mediated mitochondrial permeability transition and apoptosis in mice. Hepatology 2001; 33: 1181-1188.
Colell A, Garcia-Ruiz C, Morales A, et al. Transport of reduced glutathione in hepatic mitochondria and mitoplasts from ethanol-treated rats: effect of membrane physical properties and S-Adenosyl-L-methionine. Hepatology 1997; 26: 699-708.
Pessayre D, Mansouri A, Haouzi D, et al. Hepatotoxicity due to mitochondrial dysfunction. Cell Biol Toxicol 1999; 15: 367-373.
Susin SA, Lorenzo H, Zamzami N, et al. Molecular characterization of mitochondrial apoptosis-inducing factor. Nature 1999; 397: 441-446.
Lauterburg BH, Grattagliano I, Gmur R, et al. Noninvasive assessment of the effect of xenobiotics on mitochondrial function in human beings: studies with acetylsalicylic acid and ethanol with the use of the carbon 13-labeled ketoisocaproate breath test. J Lab Clin Med 1995; 125: 378-383.
Laskin DL. Nonparenchimal cells and hepatotoxicity. Sem Liver Dis 1990; 10: 293-304.
Gut J, Christen U, Huwyler J. Mechanisms of halotane toxicity: novel insights. Pharmacol Ther 1993; 58: 133-155.
Li J, Billiar TR. Nitric oxide. IV. Determinants of nitric oxide protection and toxicity in the liver. Am J Physiol 1999; 276: G1069-G1073.
Laskin DL, Pendino KJ. Macrophages and inflammatory mediators in tissue injury. Annu Rev Pharmacol Toxicol 1995; 35: 655-677.
Czaja MJ, Xu J, Alt E. Prevention of carbon tetrachloride-induced rat liver injury by soluble tumor necrosis factor receptor. Gastroenterology 1995; 108: 1849-1854.
Speck RF, Schranz C, Lauterburg BH. Prednisolone stimulates hepatic glutathione synthesis in mice. Protection by prednisolone against acetaminophen hepatotoxicity in vivo. J Hepatol 1993; 18: 62-67.