Table 2: Summary of subtypes and etiologies of leukocytoclastic vasculitis.12 |
|
Subtype |
n (%) |
Palpable purpura (predominant) CSVV (N = 38) |
|
Idiopathic |
29 (76.0) |
ACTDa |
2 (5.0) |
Infectionb |
6 (16.0) |
Drug reactionc |
1 (3.0) |
IgA vasculitis (N = 25) |
|
Idiopathic |
24 (96.0) |
Infectiond |
1 (4.0) |
ANCA-associated vasculitis (N = 8) |
|
Microscopic polyangiitis |
1 (12.5) |
Granulomatosis with polyangiitis |
2 (25.0) |
Eosinophilic granulomatosis with polyangiitis |
2 (25.0) |
p-ANCA, NOS |
2 (25.0) |
c-ANCA, NOS |
1 (12.5) |
Cryoglobulinemic vasculitis (N = 3) |
|
ACTDe |
1 (33.0) |
Infectionf |
2 (67.0) |
Urticaria like infection |
|
Urticarial vasculitis (N = 10) |
|
Normocomplementemic |
8 (80.0)g |
Idiopathic |
6 (75.0) |
Infectionh |
1 (12.5) |
Drug reactioni |
1 (12.5) |
Hypocomplementemic |
2 (20.0) |
ACTDj |
2 (100.0) |
ACTD = autoimmune connective tissue disease; ANCA = antineutrophil cytoplasmic antibody; c-ANCA = cytoplasmic ANCA; CSVV = cutaneous small-vessel vasculitis; NOS = not otherwise specified; p-ANCA = perinuclear ANCA. aSjögren syndrome and systemic lupus erythematosus in one patient each. bHepatitis C in two patients and hepatitis B and C, parvovirus, pneumonia, and streptococcal infection in one patient each. cAmoxicilin. dStreptococcal infection. eRheumatoid arthritis. fHepatitis C. gComplements were not checked in two patients. hHepatitis C. iEpoetin alfa. jSystemic lupus erythematosus and mixed connective tissue disease in one patient. |