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2018, Number 2

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Gac Med Mex 2018; 154 (2)

Calcificación simétrica y bilateral de ganglios basales. Serie de casos y revisión de la literatura

Jiménez-Ruiz A, Cárdenas-Sáenz O, Ruiz-Sandoval JL

Language: Spanish
References: 19
Page: 258-262
PDF size: 255.63 Kb.


ABSTRACT

Introduction: Symmetric, bilateral basal ganglia calcification is rare finding that sometimes occurs asymptomatically. Its prevalence increases with age, and the most affected site is the globus pallidus. Report of cases: A series of seven cases with clinical and imaging diagnosis of basal ganglia calcification, recorded during the 2012 to 2016 period at the Department of Internal Medicine of the Hospital Civil de Guadalajara “Fray Antonio Alcalde, is presented. Most common clinical presentation was with altered alertness, headache and seizures. There was one case with movement disorders; there were no cases identified with dementia or tetany. Conclusion: Ganglia calcification can be associated with age-related neurodegenerative changes, but it can be an initial manifestation of a variety of systemic pathologies, including disorders of the calcium metabolism, intoxication by different agents, and autoimmune and genetic diseases. Correlation of typical imaging findings with clinical manifestations and laboratory results should be established to reach a definitive judgment.


REFERENCES

  1. Rumboldt Z, Castillo M, Huang B, Huang B, editores. Brain imaging with MRI and CT: An image pattern approach. EE. UU: Cambridge University Press; 2012.

  2. Radaideh AM, Jaradat DM, Haddad FH. Prevalence of incidental basal ganglia calcification on routine brain computed tomography. RMJ. 2012;37(1) 6-8.

  3. Hathout GM. Clinical Neuroradiology. A case-based approach. Inglaterra: Cambridge University; 2009.

  4. Manyam, BV, Walters AS, Narla KR. Bilateral striopallidodentate calcinosis: clinical characteristics of patients seen in a registry. Mov Disord. 2001;16(2):258-264.

  5. Benke T, Karner E, Seppi K, Delazer M, Marksteiner J, Donnemiller E. Subacute dementia and imaging correlates in a case of Fahr’s disease. J NeurolNeurosurgPsychiatry. 2004;75(8):1163-1165.

  6. Eskandary H, Sabba M, Khajejpour F, Eskandari M. Incidental findings in brain computed tomography scans of 3000 head trauma patients Surg Neurol. 2005;63(6):550-553.

  7. Daghighi MH, Rezaei V, Zarrintan S, Pourfathi H. Intracranial physiological calcifications in adults on computed tomography in Tabriz, Iran. Folia Morphol (Warsz). 2007;66(2):115-119.

  8. Lauterbach EC, Cummings JL, Duffy J, Coffey CE, Kaufer D, Lovell M, et al. Neuropsychiatric correlates and treatment of lenticulostriatal diseases: a review of the literature and overview of research opportunities in Huntington’s, Wilson’s, and Fahr’s diseases.A report of the ANPA Committee on Research. J Neuropsychiatry Clin Neurosci. 1998;10(3):249‑266.

  9. Johnson JM, Legesse B, Camprodon JA, Murray E, Price BH. The clinical significance of bilateral basal ganglia calcification presenting with mania and delusions. J Neuropsychiatry Clin Neurosci.2013;25(1) 68-71.

  10. Abe K, Yoshimura H, Tanaka H, Fujita N, Hikita T, Sakoda S. Comparison of conventional and diffusion-weighted MRI and proton MR spectroscopy in patients with mitochondrial encephalomyopathy, lactic acidosis, and stroke-like events. Neuroradiology. 2004;46(2):113-117.

  11. Cano A, Romero AI, Bravo F, Vida JM, Espejo S. Síndrome MELAS: hallazgos neurorradiológicos. Radiología. 2002;44(2):69-74.

  12. Bianchi MC, Tosetti M, Battini R, Manca ML, Mancuso M, Cioni G, et al. Proton MR spectroscopy of mitochondrial diseases: analysis of brain metabolic abnormalities and their possible diagnostic relevance. Am J Neuroradiol. 2003;24(10):1958-1966.

  13. Basak RC. A case report of basal ganglia calcification; a rare finding of hypoparathyroidism. Oman Med J. 2009;24(3):220-222.

  14. Wong E, Dahl M. Basal Ganglia Calcification in idiopathic hypoparathyroidism. BCMJ. 2013;55(10):462-465.

  15. Rastogi R, Beauchamp NJ, Ladenson PW. Calcification of the basal ganglia in chronic hypoparathyroidism. J Clin Endocrinol Metab. 2003;88(4):1476‑1477.

  16. Mufaddel AA, Al-Hassani GA.Familial idiopathic basal ganglia calcification (Fahr`s disease).Neurosciences (Riyadh). 2014;19(3):171-177.

  17. Shahidi GA, Safdarian M. Fahr disease: idiopathic basal ganglia calcification. Iran J Neurol. 2017;16(1):53-54.

  18. Contreras P, Elso MJ, Ramírez DC, Cartier L. Pseudo-infarto cerebral como primeramanifestación de un MELAS tardío. Rev Chil Neuro-Psiquiat. 2008;46(1):35-42.

  19. Benavente E, Paira S, Roverano S, Saredo G. Encefalitis con estatus convulsivo localizado en una paciente con lupus eritematoso sistémico. Reumatol Clin. 2013;9(2):123-127.




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