2017, Number S1
Activation of the complement through lectins: role in rheumatic diseases
Language: Spanish
References: 15
Page: 231-234
PDF size: 134.34 Kb.
ABSTRACT
The complement system is activated in three pathways: classical, alternative and lectins. The lest one belongs to the innate immune system and it is the oldest in the phylogeny is still under descovering. It is vital in the defense against numerous microorganisms, but sometimes its activation produces inflammatory processes harmful to the host that cause many diseases such as some rheumatic disorders.REFERENCES
Degn SE, Jensen L, Olszowski T, Jensenius JC, Thiel S. Co-Complexes of MASP-1 and MASP-2 Associated with the Soluble Pattern-Recognition Molecules Drive Lectin Pathway Activation in a Manner Inhibitable by MAp44. J Immunol August [Internet].2013 [citado 2017 Ene 07];191(3):1334-45. Disponible en: http://www.jimmunol.org/content/191/3/1334.full
Dorta Contreras AJ, Padilla Docal B, Jensen AK, Schmitz M, Zerr I, ReiberH,Jensenius JC. "Blood- cerebrospinal fluid diffusion and intrathecal synthesis of lectin complement pathway components." Conference "IMMUNOCOLOMBIA2015 - 11th Congress of the Latin American Association of Immunology - 10mo. Congreso de la Asociación Colombiana de Alergia, Asma e Inmunología". [Internet] 2015. [citado 2017 Jun 17] Disponible en: http://www.frontiersin.org/MyFrontiers/Events/AbstractDetails.aspx?ABS_DOI=10.3389/conf.fimmu.2015.05.00022
Dorta Contreras AJ, Padilla Docal B, Pérez Martín O, Arias Morales A, HansottoReiber H, Jensenius JC. MASP2 and MBL: Dynamics and Intrathecal synthesis. Front. Immunol. Conference Abstract: 15th International Congress of Immunology (ICI). [Internet] 2013. [citado 2017 Jun 17] Disponible en: http://www.frontiersin.org/10.3389/conf.fimmu.2013.02.00929/event_abstract
González W, González-Losada C, González-Argote J, Jensenius JC. MAp44: diffusion from blood to cerebrospinal fluid and intrathecal synthesis. The FASEB Journal. [Internet] 2016. [citado 2017 Jun 17];30(1) Suppl 970.1 DOI: 10.1096/fj.1530-6860.Disponible en: http://www.fasebj.org/content/30/1_Supplement/970.1.short
Sandoval JR, Madsen HO, De Stefano G, Descailleaux-Dulanto J, Velazquez-Reinoso M, Ñique C et al. Extreme high prevalence of a defective mannose-binding lectin (MBL2) genotype in native South American West Andean populations. PLoS One [Internet] 2014. [citado 2017 Jun 17];9(10): e108943. Disponible en: http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0108943
Troldborg A, Thiel S, Jensen L, Hansen S, Laska MJ, Deleuran B et al. Collectin liver 1 and collectin kidney 1 and other complement-associated pattern recognition molecules in systemic lupus erythematosus. J Clin Exp Immunol [Internet] 2015. [citado 2017 Jun 17];182:132-8. Disponible en: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4608502/
Plawecki M, Lheritier E, Clavarino G, Jourde-Chiche N, Ouili S, Paul S et al. . Association between the Presence of Autoantibodies Targeting Ficolin-3 and Active Nephritis in Patients with Systemic Lupus Erythematosus. PLoS One [Internet] 2016. [citado 2017 Jun 17];11(9): e0160879. Disponible en: http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0160879
Osthoff M, Ngian GS, Dean MM, Nikpour M, Stevens W, Proudman S et al. Potential role of the lectin pathway of complement in the pathogenesis and disease manifestations of systemic sclerosis: a case-control and cohort study. Arthritis Res Ther [Internet] 2014. [citado 2017 Jun 17];16(6):480. Disponible en: https://arthritisresearch.biomedcentral.com/articles/10.1186/s13075-014-0480-6
Kasperkiewicz K, Eppa L, Świerzko AS, Bartłomiejczyk MA, Żuber ZM, Siniewicz-Luzeńczyk K et al. Lectin pathway factors in patients suffering from juvenile idiopathic arthritis. Immunol Cell Biol [Internet]. 2017 [citado 2017 Jun 17];95:666-75. Disponible en: https://www.nature.com/icb/journal/v95/n8/full/icb201731a.html