2011, Number 1
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Ann Hepatol 2011; 10 (1)
Arcuate ligament compression as a cause of early-onset thrombosis of the hepatic artery after liver transplantation
Vilatobá M, Zamora-Valdés D, Guerrero-Hernández M, Romero-Talamás H, Leal-Villalpando RP, Mercado MA
Language: English
References: 18
Page: 88-92
PDF size: 229.86 Kb.
ABSTRACT
Background. Early hepatic artery thrombosis (HAT) is a potentially lethal complication after orthotopic liver
transplantation (OLT) requiring immediate intervention.
Aim. To report an infrequent cause of HAT after
OLT and by itself a controversial clinical entity, the median arcuate ligament celiac artery
compression.
Case report. A 59-year-old female with hepatitis C virus-induced cirrhosis, Child B, MELD 15,
underwent cadaveric-donor OLT with complete vena cava exclusion. Type 1 hepatic artery anatomy was
found both in the donor and the recipient, the gastroduodenal artery was ligated. During the first eight
postoperative days, clinical and analytical evolution was satisfactory and Doppler ultrasound showed no abnormalities.
On the ninth postoperative day, the patient developed hypovolemic shock due to bleeding at
the hepatic artery anastomosis, surgical reconstruction was performed. Postoperative color Doppler
showed absent hepatic artery flow and an angiography suggested celiac artery compression. The patient
was explored again the same day, liberating the celiac artery from the median arcuate ligament and performing
thrombectomy and reconstruction of the hepatic artery anastomosis. The patient made a satisfactory
recovery and color Doppler showed adequate flow in the hepatic artery. She is alive, free of biliary
complications and enjoying a good quality of life 12 months after transplantation.
Conclusion. Median arcuate
ligament celiac artery compression is an infrequent anatomical variant that should be intentionally
evaluated in the recipient at the time of arterial reconstruction in OLT and specifically be considered in
early HAT to allow recognition and effective correction.
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