Díaz FA, Torres-Cárdenas Y, Fernández-Paz J, Morgado-Vega T, Díaz-Naranjo YA, Ibáñez-Montes de Oca Y

Language: Spanish
References: 11
Page: 191-196
PDF size: 42.86 Kb.

ABSTRACT

Introduction: Degenerative ataxias are the second cause of admission to our clinic, surpassed only by parkinsonism. In its treatment, there is no drug that can effectively stop the progressive course of the disease.
Objective: To evaluate treatment options for patients with degenerative ataxias by using a comprehensive, personalized and specialized rehabilitation as a tool to improve physical disability.
Material and Methods: A longitudinal study description was conducted with a sample of 25 patients admitted to our center from 2009- 2012 with a diagnosis of degenerative ataxia. The ICARS scales, Barthel Index and SF36 survey were applied before and after treatment.
Results: It was demonstrated that therapeutic intervention was more effective for the recovery of the functions related to gait, posture and kinetics as assessed by ICARS functions evaluation. All 25 patients improved these functions, changing the initial median final point from 21 to 15 in gait and posture, and from 30 to 19 points on the kinetic features. Regarding the Barthel Index, only 40% of patients had improvement in the degree of dependence, although all patients achieved higher scores showing greater independence. In all, the median scoring changed from 60 to 86 points.
Conclusions: Significant improvement was seen in most affected functions, indicating that therapeutic exercise is a mainstay in the treatment of patients with neurodegenerative diseases.

REFERENCES

1. Rodríguez GPL. Semiología neurológica. Cap. 2. La Habana: Editorial Ciencias Médicas; 2012, p. 60.

2. Cruz T, Cedeño HJ, Reynaldo R, González Y, Velázquez L. Ataxia Espinocerebelosa tipo 2: estudios predictivos y dilemas éticos asociados. Medi Sur 2010; 8. Suplemento «Resúmenes Congreso Nacional de Neurología».

3. Ruggieri VL, Arberas CL. Ataxias hereditarias. Rev Neurol 2000; 31: 288-96.

4. Espinós-Armero C, González-Cabo P, Palau-Martínez F. Ataxias cerebelosas autosómicas recesivas. Clasificación, aspectos genéticos y fisiopatología. Rev Neurol 2005; 41: 409-22.

5. Velázquez-Pérez L, Rodríguez-Labrada R, Sánchez-Cruz G, Laffita- Mesa JM, et al. Caracterización integral de la ataxia espinocerebelosa 2 en Cuba y su aplicación en proyectos de intervención. Rev Cub Salud Pública 2011; 37: 230-44.

6. Pérez-Ávila I, Fernández-Vieitez JA, Martínez-Góngora E, Ochoa- Mastrapa R, Velázquez-Manresa MG. Efectos de un programa de ejercicios físicos sobre variables neurológicas cuantitativas en pacientes con ataxia espinocerebelosa tipo 2 en estadio leve. Rev Neurol 2004; 39: 907-10.

7. Gómez-Fernández L. Plasticidad cortical y restauración de funciones neurológicas: una actualización sobre el tema. Rev Neurol 2000; 31: 749-56.

8. Bregado-Rosado JA, Almaguer-Melian W. Mecanismos celulares de la neuroplasticidad. Rev Neurol 2000; 31: 1074-5.

9. Licht S. Terapéutica por el ejercicio. La Habana: Revolucionaria; 1972.

10. Santamaría J, Tolosa ES, Valles A, Bayes A, Blesa R, Masana J. Mental depression in untreated Parkinson’s disease of recent outset. In: Yahr MD, Bergman KJ (eds.). Parkinson’s disease. Advance in Neurology. Vol. 45. New York: Raven Press;1986, p. 443-6.

11. Reynaldo-Armiñán RD, Reynaldo-Hernández R, Paneque-Herrera M, Prieto-Ávila L, Pérez-Ruiz E. Trastornos mentales en pacientes con ataxia espinocerebelosa tipo 2. Rev Neurol 2002; 35: 818-21.