medigraphic.com
ISSN 1561-3186 (Electronic)

2003, Number 1

<< Back Next >>

Rev Acta Médica 2003; 11 (1)

Nutrition and hepatic cirrhosis

Castellanos FMI

Language: Spanish
References: 29
Page:
PDF size: 83.51 Kb.


ABSTRACT

As an entity, Liver Cirrhosis (LC) is present with varying frequencies from country to country. Although its causes can be varied, alcoholic and viral factors are the most frequent ones. Malnutrition prevalence is high among cirrhotic patients because of the concurrence of a hypercatabolic state in one hand, and increased energy and nutrients requirements along with inadequate dietetic habits on the other, all of these factors difficulting nutritional replenishment. Feeding of the cirrhotic patient is dependent upon the clinical status of the liver disease, be either compensated or uncompensated (hepatic encephalopathy, digestive bleedings, ascitis, hepatorenal syndrome). In the first case, diet will be established following the principles of a healthy diet. Regarding those patients with uncompensated signs of the hepatic disease, recommendations will be set forth based on the response of the patient to the medical treatment. Energy metabolism can be used as a predictive criterion. Proper nutrition of these patients is as important as the pharmacological treatment of the disease and its complications, because allow us to perform actions oriented to improve their quality of life. Henceforth, the importante of knowing accurately the metabolic changes that ocurrs in this disease.


REFERENCES

  1. Sherlock S, Dooley J. Diseases of the liver and biliary system. 11 Edición. Blackwell Publishing Co. 2002: 377.

  2. Ginés P, Arroyo V. Cirrosis hepática. En: Farreras P, Rozman C. Medicina Interna. 13 edición. Editorial Mosby Doyma. Barcelona, 1995. Tomo 1. Volumen 1: 333-36.

  3. Reynolds TB, Kanel GC. Hepatopatía alcohólica. En: Stein JH. Medicina Interna. 2 edición. La Habana. Editorial Científico Técnica, 1987; Tomo 1. Volumen 1:228-35.

  4. Botelho de Carvalho E. Insuficiencia hepática. En: Botelho de Carvalho E. Manual de suporte nutricional. Editorial Médica e Científica Ltda. Sao Paolo. 1992:159-65.

  5. Suárez A, Díaz Y, Rodríguez M. La nutrición y el hígado. Arch Gastroenterol 1975;2:184-91.

  6. Donaghy A. Issues of malnutrition and bone disease in patient with cirrosis. J Gastroenterol Hepatol 2002;17: 462-6.

  7. Venegas LF, Holguin R, Yoza Y y cols. Evaluation and nutritional therapy in cirrhotic patients of Edgardo Regagliati Martins Hospital. Rev Gastroenterol Peru 2002;22:13-8.

  8. Dudrick SJ, Kavic SM. Hepatobiliary nutrition history and future. J Hepatolbiliary Pancreat Surg 2002; 9: 459-68.

  9. Jimenez G. Sindromes de malabsorción, fisipatología. Arch Gastroenterol 1975;2:135-59.

  10. Escarpín P, Albillos A, Barrios C, Burgos R, Figuera D, García C y cols. Hipertensión portal. JANO 1987;22: 1144-58.

  11. Bosch J. Hipertensión portal. En: Farreras P, Rozman C. Medicina Interna. 13 edición. Editorial Mosby Doyma. Barcelona. 1995; Tomo 1. Volumen 1: 288-01.

  12. Okita M, Sasagawa T, Tomioka K y cols. Habitual food intake and polyunsaturated fatty acid deficiency in liver cirrhosis. Nutrition 2002;18: 304-8.

  13. Navarro M, López H, Pérez V y cols. Selenium concentrations in serum of individuals with liver diseases cirrhosis or hepatitis: relationship with some nutritional and biochemical markers. Sci Total Environ 2002; 2291:135-41.

  14. Sherlock S, Heathcote J. Primary biliary cirrhosis. En: Hepatology (Editores: Bircher B, Benhamou B, Mc Intyre I, Rizzetto G, Rodés J). Oxford University Press. 1999. Volumen 2:1089-98.

  15. Tessari P. Protein metabolism in liver cirrhosis from albumin to muscle myofibrils. Curr Opin Nutr Metab Care 2003;6:79-85.

  16. Lin SY, Wang Y, Sep WH. Increased serum leptin concentrations correlate with soluble tumour necrosis factor receptor levels in patients with cirrhosis. Clin Endocrinol 2002; 57:805-11.

  17. Barreto J, Santana S. Soya: mitos, realidades, perspectivas. Editorial Ciencias Médicas. La Habana, 1999:41-3.

  18. Marchesini G, Bianchi G, Rosi B, Brizi M, Melchionda N. Nutritional treatment with branched chain aminoacids in dvanced liver cirrhosis. J Gastroenterol 2000;35: 7-12.

  19. Weser E, Young E. Nutrición y Medicina Interna. En: Medicina Interna (Editor: Stein JH). 2 edición. La Habana. Editorial Científico Técnica. 1987; Tomo 1. Volumen 1:289-312

  20. Gonzalo MA, Carbonero P, Aramendi T, Garcia T. Síndrome de malabsorción. Medicine 1996;7: 142- 48.

  21. Butterworth R. Complications of cirrhosis: Hepatic encephalopathy. J Gastroenterol 2000;32:171-80.

  22. Hernández EH, Valentini MC, Discalzi G. T1-weighted hypersensity in basal ganglia at brain magnetic resonance imaging are different pathologies sharing a common mechanism. Neurotoxicology 2002; 23: 669-74.

  23. Nakaya Y, Harada N, Kakui S y cols. Severe catabolic state after prolonged fasting in cirrhotic patients: effect of oral branched-chain aminoacid enriched nutrient mixture. J Gastroenterol 2002;37: 531-6.

  24. Bavdekar A, Bhave S, Pardet A. Nutrition mangement in chronic liver disease. Indian J Pediatr 2002;69: 427-31.

  25. Blendis LM, Jenkins DIA. Nutritional support in liver disease. En: Modern nutrition in health disease (Editores: Shills ME, Young VR). Séptima Edición. Philadelphia, Lea Febiger, 1988:1182-00.

  26. Sherlock S, Dooley J. Hepatic encephalopathy. En: Diseases of the liver and biliary system. 11 Edición.Blackwell Publishing Co. 2002:93-107.

  27. Garassini MA, Garassini ME. Conceptos actuales sobre ascitis. GEN 1996; 50(2):100-11

  28. Herrera Ballester A. Ascitis y su tratamiento. Europharma 1995.

  29. Tajika M, Kato M, Morí H y cols. Prognostic value of energy metabolism in patients with viral liver cirrhosis. Nutrition 2002;18: 283-4.




2020     |     www.medigraphic.com