2008, Number S1
<< Back Next >>
Rev Med UV 2008; 8 (S1)
Regulación genética de matrix metaloproteasas y sus inhibidores
Sampieri RCL
Language: Spanish
References: 21
Page: 14-17
PDF size: 105.95 Kb.
Text Extraction
No abstract
REFERENCES
Chang, C.; Werb, Z. (2001). The many faces of metalloproteases: cell growth, invasíon, angiogenesis and metastasís. Trends in Cell Biology. 11, S37-S43
Egeblad, M.; Werb, Z. (2002). New functions for the matrix metalloproteinases in cancer progression. Nature Rev. Cancer. 2, 161-174
López-Otín, C.; Overall, C.M. (2002). Protease degradomics: a new challenge for proteomics. Nature Rev. Mol. Cell Biol. 3, 509-519
Puente, X.S.; Sánchez, L.M.; Overall, C.M.; López-Otín, C. (2003). Human and mouse proteases: a comparative genomic approach. Nature Rev. Genet. 4, 544-558
Overall, C.M., López-Otín, C. (2002). Strategies for MMP inhibition in cancer: innovations for the post-trial era. Nature Rev. Cancer. 2, 657-672
Edwards, D.R., Murphy, G., Reynolds, J.J., Whitman, S.E., Docherty, A.J P., Angel, P., Heath, J.K. (1987). Transforming growth factor beta modulates the expression of collagenase and metalloproteinase inhibitor. EMBO J. 6, 1899- 1904
Edwards, D.R., Rocheleau, H., Sharma, R.R., Wills, A.J., Cowie, A., Hassell, J.A., Heath, J.K. (1992). Involvement of AP1 and PEA3 binding sites in the regulation of murine tissue inhibitor of metalloproteinases-1 (TIMP-1) transcription. Biochim. Biophys. Acta. 1171, 41-55
Baker, A.H.; Edwards, D.R.; Murphy, G. (2002). Metalloproteinase inhibitors: biological actions and therapeutic opportunities. J. Cell Sci. 115, 3719-3727
Yan, C., Boyd, D.D. 2007. Regulation of matrix metalloproteinase gene expression. J. Cell Physiol. 211, 19-26
Borden, P., Heller, R.A. 1997. Transcriptional control of matrix metalloproteinases and the tissue inhibitors of matrix metalloproteinases. Crit. Rev. Eukaryot. Gene Expr. 7, 159-178
Westermarck, J., Li, S.P., Kallunki, T., Han, J., Kähäri, V.M. 2001. p38 mitogen-activated protein kinase-dependent activation of protein phosphatases 1 and 2A inhibits MEK1 and MEK2 activity and collagenase 1 (MMP-1) gene expression. Mol. Cell. Biol. 21, 2373-2383
Vincenti, M.P., Brinckerhoff, C.E. 2002. Transcriptional regulation of collagenase (MMP- 1, MMP-13) genes in arthritis: integration of complex signaling pathways for the recruitment of gene-specific transcription factors. Arthritis Res. 4, 157-164
Reunanen, N., Li, S. P., Ahonen, M., Foschi, M., Han, J., Kähäri, V. M. 2002. Activation of p38 alpha MAPK enhances collagenase-1 (matrix metalloproteinase (MMP)-1) and stromelysin-1 (MMP-3) expression by mRNA stabilization. J. Biol. Chem. 277, 32360-32368
Hall, M.C., Young, D.A., Waters, J.G., Rowan, A.D., Chantry, A., Edwards, D.R., Clark, I.M. 2003. The comparative role of activator protein 1 and Smad factors in the regulation of Timp-1 and MMP-1 gene expression by transforming growth factor-beta 1. J. Biol. Chem. 278, 10304-10313
Cano, E., Mahadevan, L.C. 1995. Parallel signal processing among mammalian MAPKs. Trends Biochem. Sci. 20, 117-122
Pearson, G., Robinson, F., Gibson, T.B., Xu, B.E., Karandikar, M., Berman, K., Cobb M.H.2001. Mitogen-activated protein (MAP) kinase pathways: regulation and physiological functions. Endocr. Rev. 22, 153-183
Hazzalin, C.A., Mahadevan, L.C. 2002. MAPKregulated transcription: a continuously variable gene switch? Nature Rev. Mol. Cell Biol. 3, 30-40
Yang, S.H., Sharrocks A.D., Whitmarsh, A.J. 2003. Transcriptional regulation by the MAP kinase signaling cascades. Gene. 320, 3-21
Auble, D.T., Brinckerhoff, C.E. 1991. The AP-1 sequence is necessary but not sufficient for phorbol induction of collagenase in fibroblasts. Biochemistry. 30, 4629-4635
McDonnell, S.E., Kerr, L.D., Matrisian, L.M. 1990. Epidermal growth factor stimulation of stromelysin mRNA in rat fibroblasts requires induction of proto-oncogenes c-fos and c-jun and activation of protein kinase C. Mol. Cell. Biol. 10, 4284-4293
Hu, E., Mueller, E., Oliviero, S., Papaioannou, V.E., Johnson, R., Spiegelman, B.M. 1994. Targeted disruption of the c-fos gene demonstrates c-fos-dependent and -independent pathways for gene expression stimulated by growth factors or oncogenes. EMBO J. 13, 3094-3103