2007, Number 1-2
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Plasticidad y Restauración Neurológica 2007; 6 (1-2)
Guidelines of Spasticity in child with botulinum toxin type A . 500 units. Queretaro Consensum
Hernández SJ, Chio MCSMV, Arteaga RÁ, Pascual PI, Gómez HFJ, Mendoza WLS, Zorrilla J, Aguilar RF
Language: Spanish
References: 60
Page: 63-75
PDF size: 220.88 Kb.
ABSTRACT
Aims. The introduction of botulinum toxin type A (BTA) in the treatment of spasticity in children and adults was a large forward in clinical neurology in paediatric and adults patients. Currently is considered as the first choice treatment in focal spasticity.
Methods. In an attemp to achieve the optimization of this therapeutic resource we propouse these meeting specialy in the mexican country where the botulinum toxin yet is unknown invarious states. Differents clinical guidelines have been drawn up which include reviews of the evidence available about the indication of use of BTA. Spasticity is characterized by the presence of involuntary muscular hyperactivity that is often associated to pain, spams, deformity and functional disability.
Results. From the clinical point of view, the advantages of BTA are obvious
(ease of use and dosage determination, long lasting effect, reversibility should the response for better calculation of dosis or be inappropriate indication, aplications, etc.) and far outweigh its drawbacks. It can only he used after a proper selection of patients, of the therapeutic aims and of the muscular areas to be treated, and a tailor—made programme of rehabilitation must also be drawn up. Increasing experience in the use suggests that its early administration is effective in preventing or reducing the complications arisin from spasticity.
Conclusions. BTA is effective in the treatment of spasticity and plays a significant role if the clinical objectives involve functional and neuroanatomic aspects. Updated a large amount of serious-documented experience concerning its indications, effects and safety in clinical practice is already available and new information about neuronal plasticity.
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