Aldana VO, Chacón JVG, Manríquez TC, Hernández MMA, Victoria CJL, Morales BNE

Idioma: Inglés [English version]
Referencias bibliográficas: 23
Paginas: 32-37
Archivo PDF: 265.00 Kb.

RESUMEN

La trombosis multivaso es un hallazgo poco común en la angiografía coronaria. La ruptura septal ventricular es una complicación mecánica con alta mortalidad en el infarto agudo de miocardio, siendo necesario un tratamiento invasivo de emergencia. La asociación de estas entidades no ha sido reportada en la literatura médica. Este artículo presenta el caso clínico de un paciente con infarto agudo de miocardio sometido a trombólisis, coronariografía, intervención coronaria percutánea y cirugía de urgencia por trombosis coronaria simultánea de descendente anterior y coronaria derecha con rotura interventricular, choque cardiogénico refractario y muerte.

REFERENCIAS (EN ESTE ARTÍCULO)

1. Bentzon JF, Otsuka F, Virani R, Falk E. Mechanisms of plaque formation and rupture. Circ Res. 2014; 114 (12): 1852-1866.

2. Pollak PM, Parikh SV, Kizilgul M, Keeley EC. Multiple culprit arteries in patients with ST segment elevation myocardial infarction referred for primary percutaneous coronary intervention. Am J Cardiol. 2009; 104 (5): 619-623.

3. Mahmoud A, Saad M, Elgendy IY. Simultaneous multi-vessel coronary thrombosis in patients with ST-elevation myocardial infarction: a systematic review. Cardiovasc Revasc Med. 2015; 16 (3): 163-166.

4. Falk E, Shah PK, Fuster V. Coronary plaque disruption. Circulation. 1995; 92 (3): 657-671.

5. Crenshaw BS, Granger CB, Birnbaum Y, Pieper KS, Norris DC, Kleiman NS et al. Risk factors, angiographic patterns, and outcomes in patients with ventricular septal defect complicating acute myocardial infarction. GUSTO-I (Global Utilization of Streptokinase and TPA for Occluded Coronary Arteries) trial investigators. Circulation. 2000; 101 (1): 27-32.

6. Durko AP, Budde RPJ, Geleijnse ML, Kappetein AP. Recognition, assessment and management of the mechanical complications of acute myocardial infarction. Heart. 2018; 104 (14): 1216-1223.

7. Arnaoutakis GJ, Zhao Y, George TJ, Sciortino CM, McCarthy PM, Conte JV. Surgical repair of ventricular septal defect after myocardial infarction: outcomes from the Society of Thoracic Surgeons National Database. Ann Thorac Surg. 2012; 94 (2): 436-444.

8. Cinq-Mars A, Voisine P, Dagenais F, Charbonneau E, Jacques F, Kalavrouziotis J et al. Risk factors of mortality after surgical correction of ventricular septal defect following myocardial infarction: Retrospective analysis and review of the literature. Int J Cardiol. 2016; 206: 27-36.

9. Sakaguchi G, Miyata H, Motomura N, Ueki C, Fukuchi E, Yamamoto H et al. Surgical repair of post-infarction ventricular septal defect-findings from a Japanese national database. Circ J. 2019; 83: 2229-2235.

10. Schlotter F, de Waha S, Eitel I, Desch S, Fuernau G, Thiele H. Interventional post-myocardial infarction ventricular septal defect closure: a systematic review of current evidence. EuroIntervention. 2016; 12 (1): 94-102.

11. Davies MJ, Thomas A. Thrombosis and acute coronary-artery lesions in sudden cardiac ischemic death. N Engl J Med. 1984; 310 (18): 1137-1140.

12. Wang J, Cai X, Cheng X, Song P, Jiang S, Gong J. Acute myocardial infarction caused by tumor-associated thrombotic thrombocytopenic purpura: case report. Med Princ Pract. 2014; 23 (3): 289-291.

13. Chen IC, Chao TH, Tsai LM. Simultaneous anterior and inferior wall myocardial infarction in a patient with unusual coronary anatomy. Acta Cardiol Sin. 2010; 26: 119-122.

14. Yamazaki K, Funayama N, Okabayashi H, Myojo T, Gima M, Tanaka H et al. Acute coronary syndrome due to coronary thrombus formed by severe coronary spasm: a case report. J Cardiol. 2007; 50 (3): 205-212.

15. Goldstein JA, Demetriou D, Grines CL, Pica M, Shoukfeh M, ONeill WW. Multiple complex coronary plaques in patients with acute myocardial infarction. N Engl J Med. 2000; 343 (13): 915-922.

16. Asakura M, Ueda Y, Yamaguchi O, Adachi T, Hirayama A, Hori M et al. Extensive development of vulnerable plaques as a pan-coronary process in patients with myocardial infarction: an angioscopic study. J Am Coll Cardiol. 2001; 37 (5): 1284-1288.

17. Fuster V. Lewis A. Conner memorial lecture. mechanisms leading to myocardial infarction: insights from studies of vascular biology. Circulation. 1994; 90 (4): 2126-2146.

18. Ginanjar E, Yulianto Y, Salim S, Setyawan W. Double culprits in a patient with ST elevation myocardial infarction: a challenging but rewarding case. Acta Med Indones. 2018; 50 (2): 165-167.

19. Singh V, Rodríguez AP, Bhatt P, Alfonso CE, Sakhuja R, Palacios IF et al. Ventricular septal defect complicating ST-elevation myocardial infarctions: a call for action. Am J Med. 2017; 130 (7): 863.e1-863.e12.

20. Hamilton MCK, Rodrigues JCL, Martin RP, Manghat NE, Turner MS. The in vivo morphology of post-infarct ventricular septal defect and the implications for closure. JACC Cardiovasc Interv. 2017; 10 (12): 1233-1243.

21. Mubarik A, Iqbal AM. Ventricular septal rupture. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2020. Available in: https://www.ncbi.nlm.nih.gov/books/NBK534857/#article-31095.s1

22. Omar S, Morgan GL, Panchal HB, Thourani V, Rihal C, Patel R et al. Management of post-myocardial infarction ventricular septal defects: a critical assessment. J Interv Cardiol. 2018; 31 (6): 939-948.

23. Ibanez B, James S, Agewall S, Antunes MJ, Bucciarelli-Ducci C, Bueno H et al. ESC Scientific Document Group. 2017 ESC Guidelines for the management of acute myocardial infarction in patients presenting with ST-segment elevation: The Task Force for the management of acute myocardial infarction in patients presenting with ST-segment elevation of the European Society of Cardiology (ESC). Eur Heart J. 2018; 39 (2): 119-177.