2016, Número 1
<< Anterior
Ann Hepatol 2016; 15 (1)
Mice studies disentangle the role of estrogen in gallstone formation
Krawczyk M, Krasnodebski M, Krawczyk M, Lammert F
Idioma: Ingles.
Referencias bibliográficas: 14
Paginas: 141-142
Archivo PDF: 120.07 Kb.
FRAGMENTO
Sin resumen.
REFERENCIAS (EN ESTE ARTÍCULO)
Everhart JE, Ruhl CE. Burden of digestive diseases in the United States Part III: Liver, biliary tract, and pancreas. Gastroenterology 2009; 136: 1134-44.
Ko CW, Beresford SA, Schulte SJ, Matsumoto AM, Lee SP. Incidence, natural history, and risk factors for biliary sludge and stones during pregnancy. Hepatology 2005; 41: 359-65.
de Bari O, Wang TY, Liu M, Paik CN, Portincasa P, Wang DQ. Cholesterol cholelithiasis in pregnant women: pathogenesis, prevention and treatment. Annals of Hepatology 2014; 13: 728-45.
Ranelletti FO, Piantelli M, Farinon AM, Zanella E, Capelli A. Estrogen and progesterone receptors in the gallbladders from patients with gallstones. Hepatology 1991; 14: 608-12.
Koebnick C, Smith N, Black MH, Porter AH, Richie BA, Hudson S, Gililland D, et al. Pediatric obesity and gallstone disease. J Pediatr Gastroenterol Nutr 2012; 55: 328-33.
Cirillo DJ, Wallace RB, Rodabough RJ, Greenland P, LaCroix AZ, Limacher MC, Larson JC. Effect of estrogen therapy on gallbladder disease. JAMA 2005; 293: 330-9.
Wang HH, Afdhal NH, Wang DQ. Estrogen receptor alpha, but not beta, plays a major role in 17beta-estradiol-induced murine cholesterol gallstones. Gastroenterology 2004; 127: 239-49.
de Bari O, Wang HH, Portincasa P, Liu M, Wang DQ. The deletion of the estrogen receptor alpha gene reduces susceptibility to estrogen-induced cholesterol cholelithiasis in female mice. Biochim Biophys Acta 2015; 1852: 2161-9.
de Bari O, Wang TY, Liu M, Portincasa P, Wang DQ. Estrogen induces two distinct cholesterol crystallization pathways by activating ERalpha and GPR30 in female mice. J Lipid Res 2015; 56: 1691-700.
Thomas P, Dong J. Binding and activation of the seventransmembrane estrogen receptor GPR30 by environmental estrogens: a potential novel mechanism of endocrine disruption. J Steroid Biochem Mol Biol 2006; 102: 175-9.
Lammert F, Wang DQ, Hillebrandt S, Geier A, Fickert P, Trauner M, Matern S, et al. Spontaneous cholecysto- and hepatolithiasis in Mdr2-/- mice: a model for low phospholipid- associated cholelithiasis. Hepatology 2004; 39: 117-28.
Bustos BI, Pérez-Palma E, Buch S, Azócar L, Moraga C, Toilat M, Hampe J, et al. Genome-wide association study for Gallstone disease in Latin Chilean population; (Abstract 910F). Presented at the 65th Annual Meeting of The American Society of Human Genetics. October 7, 2015, in Baltimore, MD.
Lyons MA, Korstanje R, Li R, Sheehan SM, Walsh KA, Rollins JA, Carey MC, et al. Single and interacting QTLs for cholesterol gallstones revealed in an intercross between mouse strains NZB and SM. Mamm Genome 2005; 16: 152-63.
Kassi E, Spilioti E, Nasiri-Ansari N, Adamopoulos C, Moutsatsou P, Papapanagiotou A, Siasos G, et al. Vascular Inflammation and Atherosclerosis: The Role of Estrogen Receptors. Curr Med Chem 2015; 22: 2651-65.