2008, Número 3
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Rev Hosp Jua Mex 2008; 75 (3)
Vasoespasmo cerebral posterior a hemorragia subaracnoidea aneurismática: Métodos diagnósticos y escalas de valoración
Reyes SG, Mendizábal GR, Acosta GR, Ayala AA, Soto BJC, Pérez CJC, Delgado HC, Hernández ML
Idioma: Español
Referencias bibliográficas: 64
Paginas: 206-212
Archivo PDF: 199.50 Kb.
RESUMEN
El vasoespasmo cerebral como complicación de hemorragia subaracnoidea (HSA), fue descrita hace más de 50 años por Ecker
y Riemenschnaider. La incidencia mundial de hemorragia es subaracnoidea de 6 a 10.5 por cada 100,000 habitantes al año. El
vasoespasmo es causado por la presencia de sangre en el espacio subaracnoideo. El déficit neurológico causado por vasoespasmo,
rara vez aparece dentro de los tres primeros días después de la HSA, siendo más frecuente entre los días 6 8. Los aneurismas
pueden romperse en cualquier momento e incluso durante el sueño, 85% de los aneurismas rotos suele romperse hacia el
espacio subaracnoideo y 15% hacia el parénquima cerebral, ventrículos y espacio subdural. El pico de resangrado es mayor en
las primeras 24 48 horas. La utilidad del Doppler transcraneal tiene valor pronóstico en el vasoespasmo cerebral. La angiografía
cerebral sigue siendo el estándar de oro en diagnóstico de vasoespasmo. El uso de calcio antagonistas se sigue usando para
prevención de vasoespasmo cerebral.
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