2009, Número 1
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Ann Hepatol 2009; 8 (1)
Protective effect of a phytocompound on oxidative stress and DNA fragmentation against paracetamol-induced liver damage
Marotta F, Yadav H, Gumaste U, Helmy A, Jain S, Minelli E
Idioma: Español
Referencias bibliográficas: 37
Paginas: 50-56
Archivo PDF: 128.47 Kb.
FRAGMENTO
Sin resumen
REFERENCIAS (EN ESTE ARTÍCULO)
Rumack BH. Acetaminophen misconceptions. Hepatology 2004; 40, 10-15.
Lee WM. Acetaminophen and the US Acute Liver Failure Study Group: Lowering the risks of hepatic failure. Hepatology 2004; 40, 6-9.
Cohen SD, Khairallah EA. Selective protein arylation and acetaminophen-induced hepatotoxicity. Drug Metab Rev 1997; 29: 59-77.
Park BK, Kitteringham NR, Maggs JL, Pirmohamed M, Williams DP. The role of metabolic activation in drug-induced hepatotoxicity. Annu Rev Pharmacol Toxicol 2005; 45: 177-202.
Qiu Y, Benet LZ, Burlingame AL. Identification of hepatic protein targets of the reactive metabolites of the non-hepatotoxic regioisomer of acetaminophen, 3'-hydroxyacetanilide, in the mouse in vivo using two-dimensional gel electrophoresis and mass spectrometry. Adv Exp Med Biol 2001; 500: 663-673.
Knight TR, Kurtz A, Bajt ML, Hinson JA, Jaeschke H. Vascular and hepatocellular peroxynitrite formation during acetaminophen-induced liver injury: role of mitochondrial oxidant stress. Toxicol Sci 2001; 62: 212-220.
Jaeschke H, Knight TR, Bajt ML. The role of oxidant stress and reactive nitrogen species in acetaminophen hepatotoxicity. Toxicol Lett 2003; 144: 279-288.
Heinloth AN, Irwin RD, Boorman GA, Nettesheim P, Fannin RD, Sieber SO, Snell ML, Tucker CJ, Li L, Travlos GS, Vansant G, Blackshear PE, Tennant RW, Cunningham ML, Paules RS. Gene expression profiling of rat livers reveals indicators of potential adverse effects. Toxicol Sci 2004; 80: 193-202.
Knight TR, Fariss MW, Farhood A, Jaeschke H. Role of lipid peroxidation as a mechanism of liver injury after acetaminophen overdose in mice. Toxicol Sci 2003; 76: 229-236.
Kon K, Kim JS, Jaeschke H, Lemasters JJ. Increase of cytosolic ferrous iron induces the mitochondrial permeability transition in acetaminophen-induced toxicity to mouse hepatocytes (Abstract). Hepatology 2004; 40: 647A.
Reid AB, Kurten RC, McCullough SS, Brock RW, Hinson JA. Mechanisms of acetaminophen-induced hepatotoxicity: role of oxidative stress and mitochondrial permeability transition in freshly isolated mouse hepatocytes. J Pharmacol Exp Ther 2005; 312: 509-516.
Bajt ML, Knight TR, Lemasters JJ, Jaeschke H. Acetaminophen-induced oxidant stress and cell injury in cultured mouse hepatocytes: protection by N-acetyl cysteine. Toxicol Sci 2004; 80: 343-349.
Gujral JS, Knight TR, Farhood A, Bajt ML, Jaeschke H. Mode of cell death after acetaminophen overdose in mice: apoptosis or oncotic necrosis? Toxicol Sci 2002; 67: 322-328.
Ray SD, Sorge CL, Raucy JL, Corcoran GB. Early loss of large genomic DNA in vivo with accumulation of Ca2+ in the nucleus during acetaminophen-induced liver injury. Toxicol Appl Pharmacol 1990; 106: 346-351.
Shen W, Kamendulis LM, Ray SD, Corcoran GB. Acetaminophen-induced cytotoxicity in cultured mouse hepatocytes: Effects of Ca(2+)-endonuclease, DNA repair, and glutathione depletion inhibitors on DNA fragmentation and cell death. Toxicol Appl Pharmacol 1992; 112: 32-40.
Lawson JA, Fisher MA, Simmons CA, Farhood A, Jaeschke H. Inhibition of Fas receptor (CD95)-induced hepatic caspase activation and apoptosis by acetaminophen in mice. Toxicol Appl Pharmacol 1999: 156, 179-186.
Marotta F, Lorenzetti F, Harada M, Ono-Nita SK, Minelli E, Marandola P. Redox status impairment in liver and kidney of prematurely senescent mice: effectiveness of DTS phytotherapeutic compound. Ann N Y Acad Sci 2006; 1067: 408-413.
Marotta F, Harada M, Minelli E, Ono-Nita SK, Marandola P. «Accelerating aging» chemotherapy on aged animals: protective effect from nutraceutical modulation. Rejuvenation Res 2008; 11: 513-517.
Griffith OW. Determination of glutathione and glutathione disulfide using glutathione reductase and 2-vinyl pyridine. Anal Biochem 1980: 106, 207-212.
Fridovich I. Superoxide dismutases. Adv Enzymol Relat Areas Mol Biol 1974; 41: 35-97.
Beutler E. Red Cell Metabolism: A Manual of Biochemical Methods. 2nd ed. New York: Grune & Stratton, 1975. CAT activity measurement; p. 261-5.
Ohkawa H, Ohishi N, Yagi K. Assay for lipid peroxides in animal tissues by thiobarbituric acid reaction. Anal Biochem 1979; 95: 351-8.
Hjerten S, Pan H. Purification and characterization of two forms of a low affinity calcium ion-ATPase from erythrocyte membranes. Biochim Biophys Acta 1983; 728: 281-8.
Sedlack J, Lindsay RH. Estimation of total, protein bound and non sulphydryl groups in tissue with Ellman’s reagent. Anal Biochem 1968; 25: 192.
Wolozin B, Iwasaki K, Vito P, Ganjei JK, Lacanà E, Sunderland T, et al. Participation of presenilin 2 in apoptosis: enhanced basal activity conferred by an Alzheimer mutation. Science 1996; 274: 1710-1713.
Yokozawa T, Dong E. Role of ginsenoside-Rd in cisplatin-induced renal injury: Special reference to DNA fragmentation. Nephron 2001; 89: 433-438.
Lee WM, Squires RH Jr, Nyberg SL, Doo E, Hoofnagle JH. Acute liver failure: Summary of a workshop. Hepatology 2008; 47: 1401-1415.
Myers RP, Shaheen AA, Li B, Dean S, Quan H. Impact of liver disease, alcohol abuse, and unintentional ingestions on the outcomes of acetaminophen overdose. Clin Gastroenterol Hepatol 2008 (in press).
Kortsalioudaki C, Taylor RM, Cheeseman P, Bansal S, Mieli-Vergani G, Dhawan A. Safety and efficacy of N-acetylcysteine in children with non-acetaminophen-induced acute liver failure. Liver Transpl 2008; 14: 25-30.
Zorov DB, Filburn CR, Klotz LO, Zweier JL, Sollott SJ. Reactive oxygen species (ROS)-induced ROS release: a new phenomenon accompanying induction of the mitochondrial permeability transition in cardiac myocytes. J Exp Med 2000; 192: 1001-1014.
Kon K, Kim JS, Jaeschke H, Lemasters JJ. Mitochondrial permeability transition in acetaminophen-induced necrosis and apoptosis of cultured mouse hepatocytes. Hepatology 2004; 40: 1170-1179.
Powell CL, Kosyk O, Ross PK, Schoonhoven R, Boysen G, Swenberg JA, Heinloth AN, Boorman GA, Cunningham ML, Paules RS, Rusyn I. Phenotypic anchoring of acetaminophen-induced oxidative stress with gene expression profiles in rat liver. Toxicol Sci 2006; 93: 213-222.
Ray SD, Sorge CL, Raucy JL, et al. Early loss of large genomic DNA in vivo with accumulation of Ca2+ in the nucleus during acetaminophen-induced liver injury. Toxicol Appl Pharmacol 1990; 106: 346-51.
Larson AM. Acetaminophen hepatotoxicity. Clin Liver Dis 2007; 11: 525-548.
Zhang Y, Ye QF, Lu L, Xu XL, Ming YZ, Xiao JS. Panax notoginseng saponins preconditioning protects rat liver grafts from ischemia/reperfusion injury via an antiapoptotic pathway. Hepatobiliary Pancreat Dis Int 2005; 4: 207-212.
Li L, Zhang JL, Sheng YX, Guo DA, Wang Q, Guo HZ. Simultaneous quantification of six major active saponins of Panax notoginseng by high-performance liquid chromatography-UV method. J Pharm Biomed Anal 2005; 38: 45-51.
Park SA, Choi MS, Jung UJ, Kim MJ, Kim DJ, Park HM, Park YB, Lee MK. Eucommia ulmoides Oliver leaf extract increases endogenous antioxidant activity in type 2 diabetic mice. J Med Food 2006; 9: 474-479.