2007, Número S2
Rev Endocrinol Nutr 2007; 15 (S2)
Consenso en el diagnóstico y tratamiento del síndrome de Cushing
Espinosa de los Monteros-Sánchez AL, Valdivia-López J, Mendoza-Zubieta V, Mercado-Atri M, Gómez-Pérez F, Vergara-López A, Romero-Zazueta A, Hernández-García I, Reza-Albarrán A, Portocarrero-Ortiz L, Cortinas-López L, Rangel-Sánchez G, González-Villaseñor G, Martínez-Sibaja C, Orozco R
Idioma: Español
Referencias bibliográficas: 39
Paginas: 3-12
Archivo PDF: 148.06 Kb.
FRAGMENTO
INTRODUCCIÓN
El síndrome de Cushing (SC) se presenta como consecuencia de la exposición prolongada a glucocorticoides. El síndrome de Cushing exógeno, resultado de la administración de estos compuestos, es la causa más común. El SC endógeno es menos frecuente y se debe a un exceso en la producción de cortisol, como resultado de un tumor productor de ACTH, el cual puede ser de origen hipofisiario (Enfermedad de Cushing, 70% de los casos), o ectópico (15%); o bien debido a una lesión suprarrenal (adenoma, carcinoma o hiperplasia).
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